Critical Care - Volume 4, issue Suppl 1

Lorem ipsum dolor sit amet, consectetur adipiscing elit. Cras neque tellus, ornare vel pulvinar luctus, vestibulum malesuada ante. Nullam pellentesque, ipsum vel fermentum tincidunt, velit neque molestie lacus, at iaculis purus mauris in nisl. Nullam vehicula imperdiet auctor. Mauris tempus, leo eget euismod condimentum, erat justo vehicula augue, vitae ornare elit felis nec libero. Etiam et quam sit amet metus dignissim dictum sed vitae purus. Phasellus gravida hendrerit risus vitae euismod. In elementum massa facilisis nulla pulvinar at pharetra leo pellentesque.

Electronic ISSN
1466-609X
Print ISSN
1364-8535

Abstract

During the August 1998 heat wave in Tel Aviv, many patients were referred to the Tel Aviv Medical Center for acute heat-related illness. Of these, six patients were admitted to the Intensive Care Unit for severe heat stroke. We describe the clinical course during their first five days of hospital

Journal: Critical Care, vol. 4, no. Suppl 1, 2000

Abstract

Six retrospective case-control studies showed that the only difference between those patients who underwent elective infrarenal aortic aneurysm repair (IAAR) developing mesenteric traction syndrome (MTS) in the operation room (OR) and multiorgan dysfunction syndrome (MODS) during their ICU stay,

Journal: Critical Care, vol. 4, no. Suppl 1, 2000

Abstract

Heparin flush systems prevent thrombus formation on invasive monitoring devices. Unfractionated heparin increases the risk of thrombocytopenia, and drug interactions in lines. Low dose heparin, given to most patients on the unit to prevent deep vein thrombosis (DVT), decreases thrombus formation

Journal: Critical Care, vol. 4, no. Suppl 1, 2000

Abstract

Gastric mucosal PCO2 is an important parameter to evaluate the splanchnic perfusion and to estimate the prognosis of critically ill patients. Saline and gas tonometry are the existing measurement methods, with air tonometry being the golden standard. The main disadvantage of this metho

Journal: Critical Care, vol. 4, no. Suppl 1, 2000

Abstract

To determine the optimal time within the APRV phase cycle to accurately measure PaOP.

Ten consecutive patients with acute lung injury (ALI) managed with PCV and a pulmonary artery catheter (PAC) were studied. Demographic data was recorded. Patients served as their own controls and were vent

Journal: Critical Care, vol. 4, no. Suppl 1, 2000

Abstract

One of the mechanisms causing BT is impaired host defense. Metoclopramide (M) restores the depressed immune function after hemorrhage [1]. To our knowledge, there is no study investigating the effect of M on BT.

We investigated the administration of M on BT in a dog model of ischemia-reperf

Journal: Critical Care, vol. 4, no. Suppl 1, 2000

Abstract

In the Acute Respiratory Distress Syndrome (ARDS), although the edema is homogeneously distributed, a hydrostatic pressure gradient squeezes the gas out from the alveoli, causing a lung density increase along a gravity gradient. We studied by CT scan, in an oleic acid induced ARDS experimental mo

Journal: Critical Care, vol. 4, no. Suppl 1, 2000